20xx年醫(yī)學(xué)專題—低血糖顱腦損傷(參考版)
2024-11-01 17:29本頁(yè)面
【正文】 低血糖腦病的預(yù)后,第五十頁(yè)，共五十頁(yè)。li232。血糖濃度在2.78mmol/L(50mg/dl)。如果血糖低于40mg/dl(2.2mmol/L),就可能出現(xiàn)下述癥狀：。ng)總結(jié),低血糖癥相關(guān)的神經(jīng)系統(tǒng)損害。,內(nèi)容(n232。o)而死亡。u),低血糖昏迷超過(guò)6h，即會(huì)有不可逆的腦組織損害，病愈后可遺留各種腦病后遺癥嚴(yán)重者可因治療無(wú)效(w,低血糖腦病的預(yù)后(y249。)葡萄糖(2030g) 口服蔗糖 進(jìn)食物,患者(hu224。li225。,第四十七頁(yè)，共五十頁(yè)。當(dāng)然，在確診低血糖之前。 ④在相同的環(huán)境條件下，如禁食體力活動(dòng)或餐后數(shù)小時(shí)，出現(xiàn)類似的綜合性癥狀。 ②有驚厥或發(fā)作性神經(jīng)精神癥狀。ngzhu224。o)注意事項(xiàng) 對(duì)于低血糖腦病來(lái)說(shuō)，最重要的治療原則是防重于治,提高警惕及時(shí)發(fā)現(xiàn)，有效治療。,低血糖腦病的治療(zh236。j237。li225。,第四十五頁(yè)，共五十頁(yè)。x236。nz224。,第四十四頁(yè)，共五十頁(yè)。li232。,低血糖對(duì)眼(du236。)彌漫損害,Fig. 3 a–d (Patient 14) A 32yearold woman was found in a coma 2 days after she was last seen. Glucose level was 33 mg/dL at presentation. Diffusionweighted images(a, b) on the day of admission show bilaterally symmetrical confluent hyperintense lesions in the periventricular and subcortical white matters. There are also involvements of the corpus callosum and internal capsule. ADC maps (c, d) at the same levels as a and b show decreased ADC in these lesions 。,胼胝體、白質(zhì)(b225。,胼胝(pi225。zh236。nzhī)體損害,Figure 1 (A) Diffusionweighted imaging (DWI) on admission showing hyperintense lesions in the splenium of the corpus callosum and the bilateral posterior limbs of the internal capsules. (B) DWI obtained 2 h after glucose infusion showing almost full recovery except for a small part of the spleniumof the corpus callosum. (C) DWI obtained 2 days after glucose infusion showing complete regression of the hyperintense lesions.,第四十頁(yè)，共五十頁(yè)。 matrix: 128128) with increased signal intensities in bilateral corona radiata and splenium. B, Initial ADC maps with signal reduction also in bilateral corona radiata and splenium corresponding to DWI images.,第三十九頁(yè)，共五十頁(yè)。 1000 s/mm2。ngsh232。,半卵圓中心(zhōngxīn)非對(duì)稱損害,Fig. 5 a, b (Patient 17) A 91yearold diabetic man admitted for drowsiness for 10 h. Glucose level was 24 mg/dL at presentation .Diffusionweighted image (a) on the day of admission shows focal area of unilateral hyperintense lesion in the left centrum semiovale (arrows) with reduced ADC value (b),第三十八頁(yè)，共五十頁(yè)。i n225。)損害,Fig. 1 a–d (Patient 5) A 57yearold diabetic man was found in a coma 6 h after he was last seen. Glucose level was 16 mg/dL at presentation. Fluidattenuated inversion recovery image (a) on the day of admission shows slightly increased signal intensity in the cerebral cortex and basal ganglia. Diffusionweighted images (b, c) clearly show bilaterally symmetrical hyperintense lesions in the cerebr
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