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【參考文獻(xiàn)】 ?。?] Akao T, Takeyoshi I, Totsuka O, et al. Effect of the free radical scavenger MCI186 on pulmonary ischemiareperfusion injury in dogs[J]. J Heart Lung Transplant, 2006,25(8):965971. ?。?] Kimura S, Zhang GX, Nishiyama A, et al. Role of NAD(P)H oxidase and mitochondriaderived reactive oxygen species in cardioprotection of ischemic reperfusion injury by angiotensin II[J]. Hypertension, 2005,45(5):860866. ?。?] Halestrap AP, Kerr PM, Javadov S, et al. Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injury of the heart[J]. Biochim Biophys Acta,1998,1366(12):7994. [4] 唐燕華,陳朋,胡大仁,[J].江西醫(yī)學(xué)院學(xué)報,2003,43(4):2830. [5] Wang LF, Zhang HY. A theoretical investigation on DPPH radicalscavenging mechanism of edaravone[J]. Bioorg Med Chem Lett, 2003,13(21):37893792. ?。?] Abe S, Kirima K, Tsuchiya K, et al. The Reaction Rate of Edaravone 3Methyl1phenyl2pyrazolin5one(MCI186) with hydroxyl radical[J]. Chem Pharm Bull, 2004,52(2):186191. ?。?] Tsujita K, Shimomura H, Kawano H, et al. Effects of edaravone on reperfusion injury in patients with acute myocardial infarction[J].Am J Cardiol,2004,94(4):481484. ?。?] 何 蘭,唐 中,張國元,[J].臨床心血管病雜志,2005,21(1):1718. 。min)1(P)。)Μg(kg術(shù)后48