【文章內(nèi)容簡介】 000元。在受益人的項目內(nèi)，王某填寫的受益人為“法定”。 1999年 5月，其妻張某因家庭糾紛將王某殺害，王某的父母向保險公司提起索賠。保險公司經(jīng)審查，形成了兩種截然不同的意見：第一種意見認為，被保險人是被其妻故意殺害，是違法犯罪行為，不屬于意外傷害。退一步說，即使是意外傷害，也屬于免責的范疇 。第二種意見認為，被保險人被其妻故意殺害，是違法行為，但對于被保險人來說屬于意外傷害，應承擔賠償責任。你認為哪種意見正確，說明理由。 第二種意見是正確的。因為意外傷害是指外來的、突發(fā)的、非本意的、非疾病的使身體受到傷害的客觀事件。本案中，對被保險人來說，完全是一種外來的、突然的、不能預料的客觀事件，符合意外傷害的定義和特征，屬于保險公司承擔保險責任的范圍。同時，《保險法》規(guī)定：“投保人、受益人故意造成被保險人死亡、傷殘或者疾病的。保險人不承擔給付保險金的責任。投保人已交足 2年以上保險費的，保險人應當 按照合同約定向其他享有權利的受益人退還保險單的現(xiàn)金價值。受益人故意造成被保險人死亡或者傷殘的，或者故意殺害被保險人未遂的，喪失受益權?！币虼耍ｋU公司是否承擔保險責任，需要確定其妻張某是否為受益人。在《保險法》中受益人均是指定的，不存在法定受益人的概念，因此，應認定被保險人把受益人填寫為“法定”是無效的，等同于未指定。所以，其妻張某不是受益人，保險公司應承擔保險責任。但保險金應作為被保險人的遺產(chǎn)，由被保險人的繼承人申領。 3 某公司總資產(chǎn)為 4000 萬元，該公司僅將價值 1200萬元的房屋投保了火災保險，在 保險期限內(nèi)，該公司遭受了一場大火，災害造成的損失達 200萬元，其中房屋損失 186萬元，其他財產(chǎn)損失 14萬元。在救火搶險過程中發(fā)生施救費用 1萬元。分析此案例，并回答下列問題： （ 1）保險人應對該次火災事故負責賠償?shù)捻椖繎撚心男??（ 2）保險人采取什么賠償方式？ （ 3）保險人應該支付的保險賠款是多少？ （ 4）某公司應該自負的財產(chǎn)損失是多少？ 8 （ 1）保險人應對該次火災事故負責賠償?shù)捻椖繎撌欠课輷p失與部分施救費。（ 2）比例賠償方式 （ 3）保險人應該支付的保險賠款是 186萬元。 （ 4）因為 其他財產(chǎn)損失 14 萬元，所以某公司應該自負的財產(chǎn)損失是14萬元。 9 請您刪除一下內(nèi)容， O(∩ _∩ )O 謝謝?。?！ 2021 年中央電大期末復習考試小抄大全，電大期末考試必備小抄，電大考試必過小抄Acetylcholine is a neurotransmitter released from nerve endings (terminals) in both the peripheral and the central nervous systems. It is synthesized within the nerve terminal from choline, taken up from the tissue fluid into the nerve ending by a specialized transport mechanism. The enzyme necessary for this synthesis is formed in the nerve cell body and passes down the axon to its end, carried in the axoplasmic flow, the slow movement of intracellular substance (cytoplasm). Acetylcholine is stored in the nerve terminal, sequestered in small vesicles awaiting release. When a nerve action potential reaches and invades the nerve terminal, a shower of acetylcholine vesicles is released into the junction (synapse) between the nerve terminal and the ?effector‘ cell which the nerve activates. This may be another nerve cell or a muscle or gland cell. Thus electrical signals are converted to chemical signals, allowing messages to be passed between nerve cells or between nerve cells and nonnerve cells. This process is termed ?chemical neurotransmission‘ and was first demonstrated, for nerves to the heart, by the German pharmacologist Loewi in 1921. Chemical transmission involving acetylcholine is known as ?cholinergic‘. Acetylcholine acts as a transmitter between motor nerves and the fibres of skeletal muscle at all neuromuscular junctions. At this type of synapse, the nerve terminal is closely apposed to the cell membrane of a muscle fibre at the socalled motor end plate. On release, acetylcholine acts almost instantly, to cause a sequence of chemical and physical events (starting with depolarization of the motor endplate) which cause contraction of the muscle fibre. This is exactly what is required for voluntary muscles in which a rapid response to a mand is required. The action of acetylcholine is terminated rapidly, in around 10 milliseconds。 an enzyme (cholinesterase) breaks the transmitter down into choline and an acetate ion. The choline is then available for reuptake into the nerve terminal. These same principles apply to cholinergic transmission at sites other than neuromuscular junctions, although the structure of the synapses differs. In the autonomic nervous system these include nervetonerve synapses at the relay stations (ganglia) in both the sympathetic and the parasympathetic divisions, and the endings of parasympathetic nerve fibres on nonvoluntary (smooth) muscle, the heart, and glandular cells。 in response to activation of this nerve supply, smooth muscle contracts (notably in the gut), the frequency of heart beat is slowed, and glands secrete. Acetylcholine is also an important transmitter at many sites in the brain at nervetonerve synapses. To understand how acetylcholine brings about a variety of effects in different cells it is necessary to understand membrane receptors. In postsynaptic membranes (those of the cells on which the nerve fibres terminate) there are many different sorts of receptors and some are receptors for acetylcholine. These are protein molecules that react specifically with acetylcholine in a reversible fashion. It is the plex of receptor bined with acetylcholine which brings about a biophysical reaction, resulting in the response from the receptive cell. Two major types of acetylcholine receptors exist in the membranes of cells. The type in skeletal muscle is known as ?nicotinic‘。 in glands, smooth muscle, and the heart they are ?muscarinic‘。 and there are some of each type in the brain. These terms are used because nicotine mimics the act