v動(dòng)物身體圖式的模式建成i-資料下載頁(yè)
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【正文】 orter 93F reporter Lgl is required for the initial positioning of the oocyte, rather just for its maintenance at the posterior of the developing egg chambers aOrb WT Lgl is required for the initial positioning of the oocyte, rather just for its maintenance at the posterior of the developing egg chambers aOrb WT lglts3/lgl4 Lgl is required for the initial positioning of the oocyte, rather just for its maintenance at the posterior of the developing egg chambers aOrb lglts3/lgl4 WT lgl4 FRT clone Lgl functions in the oocyte positioning by affecting the DEcadherin mediated adhesion between oocyte and posterior follicle cells aArm aEgl WT Lgl functions in the oocyte positioning by affecting the DEcadherin mediated adhesion between oocyte and posterior follicle cells aArm aEgl WT lgl4 FRT clone Summary (I) Lgl is required for the establishment of the initial AP asymmetry. Lossoffunction mutations in lgl cause mispositioning of the oocyte in the anterior adjacent one of the fused egg chambers, due to lack of the stalk differentiation. Lgl is required for the formation of the oocyte polarity along the anteroposterior (AP) axis at midoogenesis aStaufen WT Lgl is required for the formation of the oocyte polarity along the anteroposterior (AP) axis at midoogenesis aStaufen WT lgl4 FRT clone Lgl is required for the formation of the oocyte polarity along the anteroposterior (AP) axis at midoogenesis aStaufen lgl4 FRT clone WT lgl4 FRT clone Lgl is required for the formation of the oocyte polarity along the dorsoventral (DV) axis at midoogenesis aGurken WT Lgl is required for the formation of the oocyte polarity along the dorsoventral (DV) axis at midoogenesis aGurken WT lglts3/lgl4 Lgl is required for the formation of the oocyte polarity along the dorsoventral (DV) axis at midoogenesis aGurken WT lglts3/lgl4 lgl4 FRT clone Inactivation of Lgl could result in a failure to reanize the oocyte MT cytoskeleton at midoogenesis aKinesin WT Inactivation of Lgl could result in a failure to reanize the oocyte MT cytoskeleton at midoogenesis aKinesin lgl4 FRT clone WT Inactivation of Lgl could result in a failure to reanize the oocyte MT cytoskeleton at midoogenesis aKinesin WT lgl4 FRT clone lgl4 FRT clone Mutations in Lgl in the posterior follicle cells lead to defects in differentiation of those mutant cells aFasciclin3 WT Mutations in Lgl in the posterior follicle cells lead to defects in differentiation of those mutant cells aFasciclin3 WT lgl4 FRT clone Defective posterior follicle cell differentiation is responsible for the oocyte polarity phenotype in lgl mutants AntibGal WT 998/12 reporter AntibGal Defective posterior follicle cell differentiation is responsible for the oocyte polarity phenotype in lgl mutants 998/12 reporter WT lgl4 FRT clone 998/12 reporter AntibGal Defective posterior follicle cell differentiation is responsible for the oocyte polarity phenotype in lgl mutants WT 998/12 reporter lgl4 FRT clone 998/12 reporter 998/12 reporter lgl4 FRT clone Summary (II) Lgl is required for the formation of the oocyte polarity along the AP and DV axes at midoogenesis. Removal of the lgl gene function from the posterior follicle cells results in a loss of the oocyte polarity that is elicited by the failure of those posterior cells to differentiate normally. Li,Q., Xin,T., Chen,W., Zhu,M., and Li,M. Cell Research. 2022, 18:372384 Current work and future direction Explore the mechanisms underlying the defects in differentiation of the stalk cells and posterior follicle cells in lgl mutants during oogenesis: Notch/Delta signaling JAK/STAT signaling EGFR signaling
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