freepeople性欧美熟妇, 色戒完整版无删减158分钟hd, 无码精品国产vα在线观看DVD, 丰满少妇伦精品无码专区在线观看,艾栗栗与纹身男宾馆3p50分钟,国产AV片在线观看,黑人与美女高潮,18岁女RAPPERDISSSUBS,国产手机在机看影片

正文內(nèi)容

細(xì)胞凋亡與衰老-閱讀頁(yè)

2024-10-14 22:13本頁(yè)面
  

【正文】 ly of the nuclear lamina and shrinkage of the nucleus. ?Proteins required for cell structure. Such as IF, actin, and gelsilin. Cleavage and inactivation of these proteins lead to changes in cell shape. Induce cell display signals marked it for phagocytosis . ?The inhibitor of CAD (Caspaseactivated Dnase, an endonuclease). Cleavage of CAD inhibitor lead to activation of CAD, once activated, CAD translocates from the cytosol to the nucleus severing DNA into fragments. ?Enzymes involved in DNA repair. Which are inactivated by caspase cleavage. DNA repair is a homeostatic activity that is inappropriate in an apoptotic cell. Molecular pathways of apoptosis Two principle pathways Extrinsic pathway Intrinsic pathway (1) Extrinsic pathway: Fas Signaling Pathway Fas (also called Apo1 or CD95) is a member of the tumor necrosis factor receptor (TNFR) superfamily. Receptormediated pathway of apoptosis Bcl2 Family(cytoplasmic factors): Bad,Bid,and bax: promote apoptosis。 and Caspase Cascade,… Activation of caspase2 is required for permeabilization of mitochondria, release of cytochrome c, and apoptosis Pathways to cell death in C. elegans and mammals
點(diǎn)擊復(fù)制文檔內(nèi)容
范文總結(jié)相關(guān)推薦
文庫(kù)吧 www.dybbs8.com
備案圖鄂ICP備17016276號(hào)-1