腸梗阻中山大學(xué)外科學(xué)-wenkub
2022-11-02 19:37:28 本頁面
【正文】 ression of obstruction. Acute and chronic obstruction Pathophysiology Motility of the small intestine The intestine contracts vigorously The patient crampy abdominal pain Finally the intestine dilated. Absorption and secretion water and electrolytes accumulate. a decrease in absorption, an increase in intestinal secretion. Infection and toxemia The bacteria proliferate, produce toxin. Vascular supply or viability promised bacteria,toxin diffuse severe peritonitis and toxemia. Shock Severe dehydration, decrease of blood volume electrolytic disturbance, acidbase imbalance, bacterial infection and toxemia, Clinical manifestations Nausea and vomiting Colicky abdominal pain Obstipation Abdominal distention. Their onset varies not only with the duration of established obstruction but also with its anatomic site. and vomiting: may be the only symptoms. 1).The nature of the vomitus. undigested food particles. bees bilious. feculent. 2).The onset and character of vomitinglevel. Recurrent vomiting of bilestained fluid (proximal ) Prolonged nausea precedes vomiting, feculent.(distal) Vomiting a late finding if the ileocecal valve prevents 2. Crampy (spastic) abdominal pain: Absent, Most prominent,paroxysmal,crampy,often diffuse, poorly localized, and lasting 13min. Between spasms, pain resolves. Borborygmus loud, coincident with cramps Severe ,continuous abdominal pain su
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