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少達(dá)到0.7mm或球部達(dá)0.9mm,325名患者,Smilde TJ, et al. Lancet. 2001。)成形術(shù)(CAS)前他汀預(yù)治療的思索,Radiology. 2006。,小結(jié)(xiǎoji233。阿托伐他汀組: LDLC,155?86mg/dL。ng)總結(jié),他汀在頸動(dòng)脈粥樣硬化中的證據(jù)。o)顯著降低CAS后30天內(nèi)心血管事件,4%,15%,P0.05,心血管事件(sh236。i)壓縮性重構(gòu)(斑塊更穩(wěn)定),210名輕微(qīngwēi)冠脈狹窄的患者,分析其管腔面積、斑塊范圍及EEM(外彈力膜)范圍,并評(píng)估重構(gòu)比率(RR),結(jié)果顯示:RR減少3.0(P0.0001),Circulation. 2006。nch233。24:175180. Kinlay S et al for the Vascular Basis Study Group. Am J Cardiol. 2002。ng 233。ng) 1875 歲 LDLC 160 mg/dL (未經(jīng)降脂治療) and 130 mg/dL (接受降脂治療) 病人接受成功的介入治療 德國(guó)8中心,常規(guī)治療*,阿托伐他汀2080 mg,131 Patients,入選人群,*常規(guī)治療包括降脂治療, 但除外阿托伐他汀 Schartl M et al. Circulation. 2001。nghu224。i)帽,血管(xu232。guǎn)重構(gòu),血管狹窄,血管閉塞,1,2,3,4,5,第三頁(yè),共三十一頁(yè)。nghu224。nɡ)分期,IMT增厚,斑塊形成(x237。,陽(yáng)性(y225。li232。njiū):德國(guó)阿托伐他汀IVUS研究(y225。88:4147.,入選患者 (n=59),第一次頸動(dòng)脈內(nèi)膜剝脫術(shù),平均治療4.5個(gè)月,第二次頸動(dòng)脈內(nèi)膜剝脫術(shù),注:研究過(guò)程中所有患者均接受標(biāo)準(zhǔn)抗血小板治療(阿司匹林100mg/d),不耐受者給予噻氯匹定 400mg/d。104:387392. Tsimikas S et al for the Myocardial Ischemia Reduction With Aggressive Cholesterol Lowering Study Investigators. Circulation. 2004。106:20552060,0.034,0.04,0.03,0.02,0.01,0,0.01,0.02,0.03,0.04,IMT的平均(p237。zhuǎn) vs 臨床大獲益,他汀逆轉(zhuǎn)斑塊的機(jī)制可能與斑塊壓縮性重構(gòu)(陰性重構(gòu))有關(guān),提示逆轉(zhuǎn)斑塊與穩(wěn)定斑塊具有一致性機(jī)制 斑塊壓縮性重構(gòu)使斑塊更穩(wěn)定,減少斑塊破裂風(fēng)險(xiǎn)(fēngxiǎn),可大幅度降低事件,Klein LW, et al. Atherosclerosis regression, vascular remodeling,and plaque stabilization.JACC. 2007,第二十三頁(yè),共三十一頁(yè)。240(1):145151,第二十六頁(yè),共三十一頁(yè)。)斑塊:強(qiáng)化他汀治療,第三十頁(yè),共三十一頁(yè)。心血管事件(sh236。,40%,